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DNA damage promotes mutations of suppressor in tumor such as calcium/calmodulin dependent serine protein kinase (CASK), p53, as well as stimulation of the epidermal growth factor receptor (EFGR) signaling pathway, which are important early events in gastric carcinogenesis (86, 87)

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In eukaryotic systems, NATs can have a bidirectional regulatory effect on the transcription of their target genes, either suppressing or enhancing the translation of the target gene’s mRNA [53, 54]