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8 Findings from current studies of APAP hepatotoxicity seem to favor the mechanism of toxic injury being related to mitochondrial and endoplasmic reticula leading to hepatocyte injury and necrosis

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This oxidative milieu persists even after glucose normalization, contributing to the phenomenon of “metabolic memory” observed in clinical trials, where early intensive glycemic control provides long-term protection against complications (Yang et al., 2024)